Objective: Acuta Aortic Dissection (AD) is a life treatenig disease with high early mortality rate.
Methods: Our thesis based on the results of our investigations, which shows that the main factor is for initiating aortic dissection is a direct development of a high energy kinetic force, generated from the interaction of pathological blood turbulences. Intramural haematoma i(IMH) is one silent, high risk appearence of the three AD-s without an entry, or re-entry which occasionally also can be observed as a complication at renal shock wave lithotripsy. This demonstrates the point, that the initiator of IMH is a high energy shock wave (high frequency resonance) which ruptures the cappillaries in the aortic wall and not the continuous low scale wall, or shear forces.
Results: The above phenomenon is a blood mass-kinetic energy interaction having a relattive negativ vector. It rise in, elevates the vessel wall in the two other AD form, either the intima, the media or the entire aortic wall as well. The above pathophysiology might derive from the fact that the wall of the mammalian vascular system has a natural "Given Nature" feture to tolerate extreme high blood pressure/10x the normal/ from inside-out only i.e.:multiple G-forces at military space pilots, Valsalve Manouver at professional weightlifter, but cannot tolerate significant high scale forces affecting it opposite direction.
Conclusion: A symplified mathematical formula:Kinetic force vector devided by aortic wall resistance allow us to estimate the correct prognosis for either a local intimal injury only, or for a complete acut aortic dissection.